N6-methyladenosine (m6A) modification is a common form of RNA modification, which has attracted widespread attention in the study of preeclampsia in recent years. This review summarizes the role mechanism of m6A modification in preeclampsia and its complications from the aspects of m6A methyltransferase, demethylase, and recognition proteins. Research shows that the abnormal expression of m6A modification-related genes and proteins is closely related to the onset of preeclampsia, which may promote the occurrence and development of preeclampsia by affecting placental development, inflammatory response, and other processes. m6A modification may also be involved in the pathogenesis of common complications of preeclampsia such as cardiovascular diseases, and kidney and liver damage. Although current research has initially revealed the role of m6A modification in preeclampsia, its precise mechanism and therapeutic potential still need further study. Future research is expected to provide a theoretical basis for the new diagnosis and treatment methods of preeclampsia